Select Subject

All (8760)

Agronomy (158)

Animal Husbandry,... (64)

Aquatic Science (16)

Astronomy (36)

Aviation (22)

Basic Medicine (184)

Biology (504)

Chemical Engineering (103)

Chemistry (635)

Civil and Archite... (105)

Clinical Medicine (236)

Computer Science ... (924)

Dynamic and Elect... (121)

Earth Science (119)

Economics (67)

Education (31)

Electrics, Commun... (927)

Energy Science an... (67)

Engineering and T... (15)

Environmental Sci... (140)

Food Science and ... (63)

Forestry (36)

Hydraulic Enginee... (40)

Mechanics (240)

Metallurgical Eng... (32)

Mine Engineering ... (38)

Nuclear Science a... (21)

Pharmacy (98)

Physics (920)

Preventive Medici... (37)

Psychology (20)

Security Science ... (17)

Sports Science (6)

Surveying and Map... (15)

Textile Science a... (16)

Traditional Chine... (56)

Transportation En... (57)

Select/Unselect all

For Selected Papers

Saved Papers

Please enter a name for this paper to be shown in your personalized Saved Papers list


	1. Pathological lesion and the antigenic localization of highly pathogenic avian influenza virus in Sheldrake Ducks
	LIU Xiaojing,ZHENG Shimin,SUN Zhengyang,GAO Xueli,LIU Chaonan
	Animal Husbandry, Veterinary Medicine 14 May 2017
	Show/Hide Abstract \| Cite this paper︱Full-text: PDF (0 B)

	Abstract:In this paper, to determine the pathogenicity and antigen distribution of China early H5N1 highly pathogenic avian influenza virus (HPAIV) A/GS/LN/45/04, the Sheldrake ducks were inoculated via the nostrils. Infected ducks appeared depression, orticollis and nystagmus clinical signs, hematoxylin and eosin (HE) stain found multiple organs showed different degrees of pathological changes. In addition, by Immuno-histochemistry (IHC) stain found that A/GS/LN/45/04 virus could duplicated in multiple organs, especially in brain and pancreas. These results could provide some important theoretical basis for the further study of Chinese AIVs.
	TO cite this article:LIU Xiaojing,ZHENG Shimin,SUN Zhengyang, et al. Pathological lesion and the antigenic localization of highly pathogenic avian influenza virus in Sheldrake Ducks[OL].[14 May 2017] http://en.paper.edu.cn/en_releasepaper/content/4733275


	2. Effects of Bioefficacy of DL-2-Hydroxy-4-Methylthio-Butanoic Acid on Egg Production and Egg Quality in Laying Hens Fed on Medium or Low Nutrient Scheme
	Jianmei Wan,Jianping Wang,Xuemei Ding,Shiping Bai,Qiufeng Zeng,Huanwei Peng,Zhuowei Su,Yue Xuan,Keying Zhang
	Animal Husbandry, Veterinary Medicine 21 April 2017
	Show/Hide Abstract \| Cite this paper︱Full-text: PDF (0 B)

	Abstract:Two experiments were carried out to investigate the effects of bioefficacy of DL-2-Hydroxy-4-Methylthio-Butanoic Acid (HMTBA) for layers fed on medium or low nutrient level diets under restricted feeding (Exp.1) or ad libitum feeding (Exp.2) on egg production and quality. In Exp.1, a total of 2000 layers were assigned to 10 treatments in a completely randomized design, with 10 replicates per treatment, whilst in Exp.2 there were 10 replicates (15 hens per replicate) of 10 treatments. In all experiments, the bioefficacy of HMTBA was set at 80%, 84%, and 88% (on weight basis). In Exp.1, Methionine deficiency significantly (P < 0.05) reduced egg weight and eggshell weight regardless of the dietary nutrient level. Bioefficacy of HMTBA had no influence (P > 0.05) on any egg production or egg quality parameters of hens when compared with DL-methionine (DLM) treatments. In Exp.2, egg production, ADFI, egg weight, egg mass and feed conversion ratio significantly declined due to methionine deficiency (P < 0.01). There was no difference (P > 0.05) between HMTBA and DLM on egg production and egg quality parameters within each dietary nutrient level. In conclusion, bioefficacy of HMTBA can be set at 88% on weight basis regardless of dietary nutrient level.
	TO cite this article:Jianmei Wan,Jianping Wang,Xuemei Ding, et al. Effects of Bioefficacy of DL-2-Hydroxy-4-Methylthio-Butanoic Acid on Egg Production and Egg Quality in Laying Hens Fed on Medium or Low Nutrient Scheme[OL].[21 April 2017] http://en.paper.edu.cn/en_releasepaper/content/4727788


	3. Effects of methionine source and level on hepatic methionine and lipids metabolism in laying hens fed with low or moderate nutrient level diet under restricted feeding
	Jianmei Wan,Jianping Wang,Xuemei Ding,Shiping Bai,Qiufeng Zeng,Huanwei Peng,Zhuowei Su,Yue Xuan,Keying Zhang
	Animal Husbandry, Veterinary Medicine 20 April 2017
	Show/Hide Abstract \| Cite this paper︱Full-text: PDF (0 B)

	Abstract:The study was carried out to investigate the effects of methionine source and level on hepatic methionine and lipid metabolism of commercial laying hens fed with low nutrient level (LN) or moderate nutrient level (MN) diets under restricted feeding. A completely randomized design was used. Within each nutrient level, there were 2 methionine sources (DL-methionine, DLM; DL-2-hydroxyl-4-methylthio-butiric acid, HMTBA) and 3 supplementation levels (LN: 0, 0.091 and 1%; MN: 0, 0.113 and 1%). A total of 2000 layers was allocated to 10 dietary treatments with 10 replicates for each. For hens fed LN diets: regardless of nutrient level, methionine source and level had no effects on concentration of Hcy in liver or plasma; hens treated with DLM had higher plasma Met and Tau concentration than those treated with HMTBA; when compared with the control group, adding methionine (HMTBA source) at 0.091% or 1% markedly increased the expression of GNMT; adding methionine (DLM source) at 1% significantly increased the expression of BHMT. Under the condition of feeding MN diets: plasma Met concentration significantly increased with methionine supplementation. Adding methionine (HMTBA source) at 0.113% or 1% significantly increased the expression of ADA and adding methionine (HMTBA source) at 0.113% significantly increased the expression of MS. When control diet was fed, hens fed LN diets had higher expression of BHMT and MS. When adding methionine (HMTBA source) at 1%, hens fed LN diets had higher expression of SAHH, MTHFR, CEBPβ, AdipoR2, INF-α and IL-1β. When adding methionine (DLM source) at 1%, feeding LN diet significantly increased the expression of TNF-α. In conclusion, methionine deficiency may increase the methionine regeneration by up-regulating the expression of genes involved in hepatic methionine metabolism. Under the condition of adding excess HMTBA, feeding low-nutrient diet may increase the folate-dependent pathway of methionine regeneration, SAMe-dependent transmethylation, fat synthesis and inflammatory cytokines in liver, and increase the potential risk of liver diseases.
	TO cite this article:Jianmei Wan,Jianping Wang,Xuemei Ding, et al. Effects of methionine source and level on hepatic methionine and lipids metabolism in laying hens fed with low or moderate nutrient level diet under restricted feeding[OL].[20 April 2017] http://en.paper.edu.cn/en_releasepaper/content/4727428


	4. The relative bio-efficacy of DL-methionine and DL-methionine hydroxyanalogue calcium saltcompared to L-methion

Alert Name:
Alerting to:
Authentication email will be sent to your email address in 24 hours
Frequency:
Email Message Format:	Plain text Graphical(HTML)

Complete the form below and we will recommend the selected titles to your friends on your behalf. * Indicates a required field.
Your name*:
Your email address*:
Recipient's name*:
Recipient's email address*:
(multiple recipient's names and email addresses should be separated with semicolons)
Your comments:	I thought you would find the page(s) useful.

Your name:
Your email address:
Recipient's name:
Recipient's email address:
(multiple recipient's names and email addresses should be separated with semicolons)
Your comments:	I thought you would find this page useful.

Disclaimer: This message was sent to your friend using the "Send it to a friend" facility on the Sciencepaper Online’ WWW site, http://www.paper.edu.cn/en. The Sciencepaper Online is not responsible for the content of this email, and anything said in this email does not necessarily reflect the Sciencepaper Online's views.

	Check out RSS, or use RSS reader to subscribe this item