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Does FcγRIII (CD16) mediate modified C-reactive protein-induced endothelial cells dysfunction?
Ji Shangrong *
Lanzhou Univeristy
*Correspondence author
#Submitted by
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Funding: 教育部博士点基金,教育部博士点基金（No.20050730028,20070730015）
Opened online:10 September 2008
Accepted by: none
Citation: Ji Shangrong.Does FcγRIII (CD16) mediate modified C-reactive protein-induced endothelial cells dysfunction?[OL]. [10 September 2008] http://en.paper.edu.cn/en_releasepaper/content/23936
 
 
Atherosclerosis and its complications, in particular acute coronary heart disease (ACHD) is the single leading cause of morbidity and mortality world wide. Among the clinically useful risk markers for development of ACHD, the classical acute-phase reactant C-reactive protein (CRP) has received considerable attention. Accumulating evidence indicates that CRP is not merely a corollary marker but rather an actual mediator/modulator of the inflammation that contributes to ACHD.
Keywords:CD16, C-reactive protein, Atherosclerosis
 
 
 

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