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1. Perspectives of traditional Chinese medicine in pancreas protection for acute pancreatitis | |||
LI Jun,ZHANG Shu,ZHOU Rui,ZHANG Jian,LI Zongfang | |||
Clinical Medicine 10 May 2017 | |||
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Abstract:Acute pancreatitis (AP) is one of the most common diseases. AP is associated with significant morbidity and mortality, but it lacks specific and effective therapies. Traditional Chinese medicine (TCM) is one of the most popular complementary and alternative medicine modalities worldwide for the treatment of AP. The current evidence from basic research and clinical studies has shown that TCM has good therapeutic effects on AP. This review summarizes the widely used formulas, single herbs and monomers that are used to treat AP and the potential underlying mechanisms of TCM. Because of the abundance, low cost, and safety of TCM as well as its ability to target various aspects of the pathogenesis, TCM provides potential clinical benefits and a new avenue with tremendous potential for the future treatment of AP. | |||
TO cite this article:LI Jun,ZHANG Shu,ZHOU Rui, et al. Perspectives of traditional Chinese medicine in pancreas protection for acute pancreatitis[J]. |
2. Different Treatment to AAST Grade III Blunt Pancreatic Trauma According to Injured Site | |||
Kong Wencheng | |||
Clinical Medicine 18 November 2014 | |||
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Abstract:To determin if simple drainage operation is optimal to American Association for the Surgery of Trauma (AAST) grade III pancreatic trauma. Methodology: A series of 23 AAST grade III patients of blunt pancreatic trauma was retrospectively reviewed. A standardized operation of simple drainage was performed. Morbidity was compared between neck injured group and body/tail injured group. Results: The late-stage reoperation rate for unresolved pancreatic fistula was significantly lower in neck injury group than in body/tail injury group. Conclusions: Different management should be adopted according to the injured site within grade III trauma. Simple drainage surgery should be extended from grade IV/V injury to include neck injury of grade III trauma and distal pancreatectomy can be restricted to body or tail injury. | |||
TO cite this article:Kong Wencheng. Different Treatment to AAST Grade III Blunt Pancreatic Trauma According to Injured Site[OL].[18 November 2014] http://en.paper.edu.cn/en_releasepaper/content/4618882 |
3. Different expression of cytokine in spleen tissue and macrophage in cirrhotic patients with hypersplenism | |||
Li Zongfang,Li Aimin,Ma Shangyu,Su Qinghua,Zhang Shu,Liu Xiaogong | |||
Clinical Medicine 31 March 2008 | |||
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Abstract:Purpose: Hepatitis B virus re-infection is a difficult problem to manage after liver transplantation (LT) in patients with cirrhosis. Whether performing a splenectomy at the time of LT would be beneficial or not remains controversial. This study was designed to investigate the functional changes of splenic macrophages in cirrhotic patients with hypersplenism in order to provide additional evidences by which to assess the value of splenectomy. Methods: Fourteen cirrhotic patients with hypersplenism and six controls were enrolled in the study. Serum lipopolysaccharide (LPS) was detected with a limulus assay. The differential expression of cytokines by splenic tissue and splenic macrophages between the cirrhosis and control groups was compared with cytokine arrays. Furthermore, splenic macrophages were cultured and stimulated with LPS, after which tumor necrosis factor (TNF)-α and interleukin (IL)-12 levels in the supernatant were determined. Results: In cirrhotic patients, serum LPS levels increased significantly. Interferon (IFN)-γ, TNF-β, and transforming growth factor (TGF)-β upregulated, whereas IL-4 and IL-5 levels didn’t change in splenic tissue. TNF-α upregulated significantly, while IL-4 and IL-5 levels had no significant changes in splenic macrophages. The IL-12 levels in culture media of splenic macrophages from cirrhotic patients were significantly lower than in controls after LPS stimulation. Conclusion: Endotoxemia and predominant Th1 inflammation in splenic tissue caused splenic macrophage M1 activation in cirrhotic patients with hypersplenism, but the immune functions of splenic macrophages were impaired. | |||
TO cite this article:Li Zongfang,Li Aimin,Ma Shangyu, et al. Different expression of cytokine in spleen tissue and macrophage in cirrhotic patients with hypersplenism[OL].[31 March 2008] http://en.paper.edu.cn/en_releasepaper/content/19890 |
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